Benign paroxysmal positional vertigo

Last updated: March 10, 2022

Summarytoggle arrow icon

Benign paroxysmal positional vertigo (BPPV) is a common disorder of the inner ear thought to be caused primarily by otoconia (canaliths) dislodging and migrating into one of the semicircular canals, most commonly the posterior semicircular canal, where it disrupts the endolymph dynamics. BPPV is the most common cause of peripheral vertigo. The primary symptom of BPPV is episodic vertigo that lasts < 1 minute, triggered by sudden changes in head posture in relation to gravity (e.g., bending forwards, rapidly standing up). BPPV is a clinical diagnosis that is supported by a combination of characteristic features as well as the presence of nystagmus and vertigo elicited by provoking maneuvers (e.g., Dix-Hallpike test). Canalith repositioning maneuvers (CRM), such as the Epley maneuver, are the preferred treatment of BPPV. Vestibular rehabilitation may be used as an adjunct to (CRM). Vestibular suppressants and surgery are reserved for patients with intractable or severely-disabling BPPV.

See also “Vertigo.”

Definitiontoggle arrow icon

Episodic vertigo triggered by certain changes in the position of the head [1]

Classificationtoggle arrow icon

BPPV can be classified into the following subtypes depending on which semicircular canal is involved.

Classification of BPPV [1]


Definition and epidemiology Provoking maneuvers
(Diagnostic maneuvers)
Posterior semicircular canal BPPV

Lateral semicircular canal BPPV

Anterior semicircular canal BPPV [3][4]

Epidemiologytoggle arrow icon

  • Prevalence
    • BPPV is the most common type of peripheral vestibular vertigo with a prevalence of ∼ 2%. [5]
    • BPPV is the underlying cause in approx. 40% of geriatric patients presenting with dizziness. [1]
  • Sex: > [1][5]
  • Age: peak incidence between 50–60 years [5]

Epidemiological data refers to the US, unless otherwise specified.

Etiologytoggle arrow icon

Although the exact etiology is unknown in most patients with BPPV, the condition is thought to result from the dislodgement or abnormal adherence of otoconia (see “Pathophysiology” section). The etiology of dislodged or adherent otoconia is described here.

Pathophysiologytoggle arrow icon

Although incompletely understood, BPPV is thought to occur due to dislodged or abnormally adherent otoconia, causing semicircular canal dysfunction. [1][2]

Clinical featurestoggle arrow icon

  • Episodic vertigo (spinning sensation) [1][2]
    • Sudden (“paroxysmal”) and recurrent episodes
    • Lasts several seconds (typically ≤ 1 minute)
    • Triggered by certain head movements (positional vertigo) after a latency of a few seconds.
    • Associated with:
  • Triggers: Quick rotation of the head relative to gravity is the main trigger of BPPV (see ''Pathophysiology'' section). [1][2]
    • Lying down, reclining, or standing up quickly [5]
    • Rolling over in bed
    • Bending forwards
    • Suddenly jerking the head to look up or down
  • Provoking maneuvers for BPPV: See “Diagnostics” section.

BPPV does not typically cause cochlear (e.g., hearing loss or tinnitus) or neurological symptoms.

Diagnosticstoggle arrow icon

Approach [1][5]

The following focuses on patients with triggered episodic vestibular syndrome; see “Approach to vertigo” for details on clinical evaluation, targeted testing (e.g., HINTS examination), and neuroimaging for patients with undifferentiated acute vestibular syndrome.

Diagnostic criteria for posterior semicircular canal BPPV [1]

All of the following criteria should be met to confirm the clinical diagnosis of posterior canal BPPV. [5]

Provoking maneuvers for BPPV [1][5]

  • Definition: set of specific sequential maneuvers used to provoke symptoms in an individual with suspected BPPV
  • Findings: Positional vertigo associated with nystagmus triggered on specific maneuvers is considered a positive test and is diagnostic of BPPV.
    • The following characteristics of nystagmus should be noted:
      • Latency: typically 2–5 seconds
      • Direction: The direction of nystagmus indicates which specific canal is affected (see individual maneuvers below for more details).
      • Duration: < 1 minute (typically 30 seconds) [5]
      • Reversal: The pattern of nystagmus reverses direction when the patient is made to sit in a neutral position at the end of the provoking maneuver.
      • Fatigability: Intensity of nystagmus decreases on repeated sequential testing (not recommended). [1]
  • Approach

Dix-Hallpike maneuver [1][7]

Supine head roll test [1][5]

Additional diagnostic evaluation [1]

Audiometry [1]

  • Indication: suspected hearing loss
  • Supportive findings: typically normal in BPPV

Vestibular function tests [1]

  • Definition: tests to identify nystagmus in response to vestibular stimulation and thereby assess the integrity and function of the vestibular apparatus of the inner ear
  • Indications
    • BPPV with atypical nystagmus
    • Suspected concomitant vestibular pathology [1]
    • Refractory BPPV
  • Supportive findings: Vestibular function is typically normal in BPPV.

Neuroimaging [8]

  • Modalities
    • MRI head and internal auditory canal with and without IV contrast
    • CT temporal bone without IV contrast
  • Indications [1][8]
    • Suspected BPPV with atypical features, such as:
    • To rule out intracranial causes of vertigo
    • Preoperative planning for refractory BPPV
  • Supportive findings: normal in BPPV [1]

Differential diagnosestoggle arrow icon

The differential diagnoses listed here are not exhaustive.

Treatmenttoggle arrow icon

Although BPPV is often considered a condition that resolves spontaneously, canalith repositioning maneuvers (CRM) are the preferred first-line treatment. Vestibular rehabilitation therapy may be useful as an adjunct to CRM; both forms of therapy can be performed at home, by patients themselves. Patients with refractory symptoms or atypical symptoms should be evaluated for alternative diagnoses. [1][5]

Canalith repositioning maneuvers (CRM) [1]

  • Definition: set of specific sequential maneuvers performed to mobilize the otoconia out of the involved semicircular canal and back into the vestibule
  • Indication: first-line treatment for BPPV
  • General considerations
  • Outcome
    • Up to 80–90% success rate [6][11]
    • Recurrences requiring repeat CRM are expected. [5][12]
    • Canal conversion

Epley maneuver [1][5]

  • Indication
  • Procedure
    1. The initial steps are the same as those of the Dix-Hallpike maneuver
      • Ask the patient to sit upright on the examination table and to keep their eyes open during the procedure.
      • Rotate the head by 45° towards the affected side.
      • Keeping the neck rotated, quickly lay the patient in a supine position with the neck slightly extended (approx 20°) and the affected ear facing down at 45°
    2. Hold this position for 30 seconds or until the resolution of nystagmus.
    3. Turn the patient's head by 90° toward the unaffected side; hold this position for 30 seconds or until resolution of nystagmus.
    4. Turn the patient's head and body by 90° towards the unaffected side such that the patient is now lying on their side with their face turned toward the ground.
    5. Hold this position for 30 seconds or until the resolution of nystagmus.
    6. Bring the patient back to a seated, upright position with the head in the neutral position.
    7. After completion of the maneuver, ask the patient to remain in this position for about 15 minutes.

Vestibular rehabilitation therapy [1]

Vestibular rehabilitation refers to different physical exercises to treat dizziness and balance disorders that the patients perform either at home or with the help of a clinician.

  • Indications
    • Adjunctive therapy to CRM [1]
    • Refractory BPPV
    • Patient refusal or contraindications to CRM
    • Patients with preexisting conditions may benefit the most.
  • Goal: induce vestibular habituation or adaptation to gravitational changes to minimize the frequency of episodic vertigo
  • Options
    • There are several exercises for vestibular rehabilitation (e.g., Brandt-Daroff exercise, Cawthorne-Cooksey exercise) that are especially helpful for BPPV.
    • Exercises may be customized by a trained specialist

Observation (watchful waiting) [1]

  • Indication [6]
    • Mild, infrequent symptoms
    • Relative contraindications to CRM
    • History of complications from prior CRM
    • Patient refusal of CRM
  • Relative contraindication: patients at high risk of falls or severe injury from falls [6]
  • Procedure
    • Withhold CRM and vestibular rehabilitation
    • Advise patients to avoid postures or activities that trigger symptoms (see ''Triggers'' in ''Clinical Features” section).
    • Reassess patients after 1 month

Medical therapy (vestibular suppressants) [1]

Vestibular suppressants are not routinely indicated due to their adverse effect profile and potential of suppressing the central vestibular compensatory system. Patients should be counseled about their potential side effects.

  • Indications
    • Symptomatic control during an acute attack (rare)
    • Intractable BPPV
    • Patients who refuse to undergo canalith repositioning maneuvers
    • Before provoking or repositioning maneuvers
  • Options: See “Vestibular suppressants”.
  • Duration of therapy
    • As short a period as possible
    • Acute attacks or premaneuver prophylaxis: One-time dosing may be adequate.
    • In patients with severe intractable BPPV: variable; 2–6 months [13]

Chronic use of vestibular suppressants is contraindicated in BPPV because it can inhibit central compensation and potentially exacerbate chronic gait and postural instability. [1]

Surgical treatment [2]

Complicationstoggle arrow icon

We list the most important complications. The selection is not exhaustive.

Prognosistoggle arrow icon

  • Spontaneous resolution (with observation alone) [2][6]
    • Approx. 25% at 1 month
    • Approx. 50% at 3 months
  • Recurrence rate: annually around 15% [5]

Referencestoggle arrow icon

  1. Bhattacharyya N, Gubbels SP, Schwartz SR, et al. Clinical Practice Guideline: Benign Paroxysmal Positional Vertigo (Update). Otolaryngol Head Neck Surg. 2017; 156 (3_suppl): p.S1-S47.doi: 10.1177/0194599816689667 . | Open in Read by QxMD
  2. You P, Instrum R, Parnes L. Benign paroxysmal positional vertigo. Laryngoscope Investig Otolaryngol. 2018.doi: 10.1002/lio2.230 . | Open in Read by QxMD
  3. Califano L, Salafia F, Mazzone S, Melillo MG, Califano M. Anterior canal BPPV and apogeotropic posterior canal BPPV: two rare forms of vertical canalolithiasis.. Acta Otorhinolaryngol Ital. 2014; 34 (3): p.189-97.
  4. Anagnostou E, Kouzi I, Spengos K. Diagnosis and Treatment of Anterior-Canal Benign Paroxysmal Positional Vertigo: A Systematic Review.. Journal of clinical neurology (Seoul, Korea). 2015; 11 (3): p.262-7.doi: 10.3988/jcn.2015.11.3.262 . | Open in Read by QxMD
  5. Kim J-S, Zee DS. Benign Paroxysmal Positional Vertigo. N Engl J Med. 2014; 370 (12): p.1138-1147.doi: 10.1056/nejmcp1309481 . | Open in Read by QxMD
  6. Bhattacharyya N, Baugh RF, Orvidas L, et al. Clinical practice guideline: benign paroxysmal positional vertigo.. Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery. 2008; 139 (5 Suppl 4): p.S47-81.doi: 10.1016/j.otohns.2008.08.022 . | Open in Read by QxMD
  7. Nguyen-Huynh AT. Evidence-based practice: management of vertigo.. Otolaryngol Clin North Am. 2012; 45 (5): p.925-40.doi: 10.1016/j.otc.2012.06.001 . | Open in Read by QxMD
  8. Sharma A, Kirsch CFE, Aulino JM, et al. ACR Appropriateness Criteria® Hearing Loss and/or Vertigo. J Am Coll Radiol. 2018; 15 (11): p.S321-S331.doi: 10.1016/j.jacr.2018.09.020 . | Open in Read by QxMD
  9. Ballve Moreno JL, Carrillo Muñoz R, Villar Balboa I, et al. Effectiveness of the Epley's maneuver performed in primary care to treat posterior canal benign paroxysmal positional vertigo: study protocol for a randomized controlled trial.. Trials. 2014; 15: p.179.doi: 10.1186/1745-6215-15-179 . | Open in Read by QxMD
  10. Cranfield S, Mackenzie I, Gabbay M. Can GPs diagnose benign paroxysmal positional vertigo and does the Epley manoeuvre work in primary care?. Br J Gen Pract. 2010; 60 (578): p.698-9.doi: 10.3399/bjgp10X515557 . | Open in Read by QxMD
  11. Helminski JO. Effectiveness of the Canalith Repositioning Procedure in the Treatment of Benign Paroxysmal Positional Vertigo. Phys Ther. 2014; 94 (10): p.1373-1382.doi: 10.2522/ptj.20130239 . | Open in Read by QxMD
  12. Newman-Toker D. Emergency Neuro-Otology - Diagnosis and Management of Acute Dizziness and Vertigo. Elsevier Health Sciences ; 2015
  13. Ganança MM, Caovilla HH, Ganança FF, et al. Clonazepam in the pharmacological treatment of vertigo and tinnitus.. Int Tinnitus J. 2002; 8 (1): p.50-3.
  14. Barton JJ. Benign paroxysmal positional vertigo. In: Post TW, ed. UpToDate. Waltham, MA: UpToDate. Last updated: January 8, 2016. Accessed: April 3, 2017.
  15. Li JC. Benign Paroxysmal Positional Vertigo. Benign Paroxysmal Positional Vertigo. New York, NY: WebMD. Updated: March 9, 2017. Accessed: April 3, 2017.
  16. Basura GJ, Adams ME, Monfared A, et al. Clinical Practice Guideline: Ménière’s Disease. Otolaryngol Head Neck Surg. 2020; 162 (2_suppl): p.S1-S55.doi: 10.1177/0194599820909438 . | Open in Read by QxMD
  17. Kao WT, Parnes LS, Chole RA. Otoconia and otolithic membrane fragments within the posterior semicircular canal in benign paroxysmal positional vertigo.. Laryngoscope. 2017; 127 (3): p.709-714.doi: 10.1002/lary.26115 . | Open in Read by QxMD
  18. Güneri EA, Çakır A, Mutlu B. Validity and Reliability of the Diagnostic Tests for Ménière's Disease.. Turkish archives of otorhinolaryngology. 2016; 54 (3): p.124-130.doi: 10.5152/tao.2016.1697 . | Open in Read by QxMD

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